AXIOMPHARMACEUTICALS
Research Use Only18+Not for Human ConsumptionReconstitution Materials Not Supplied
Peptide Database

Cognition & neuromodulation

PE-22-28 (Mini-Spadin)

TREK-1 Channel Blocker · Shortened Spadin Analog

Research use onlyGBP reference pricing where listedUK dispatch on catalogue items

Overview

Synthetic heptapeptide derived from Spadin positions 22-28, functioning as potent TREK-1 antagonist with selectivity and duration versus parent compound. Primary research focus on rapid antidepressant effects.

Selectively blocks TREK-1 potassium channels (IC50: 0.12 nM). serotonin neurotransmission in dorsal raphe nucleus, triggering CREB activation and hippocampal neurogenesis.

Primary research focus with rapid effects in behavioral models within 4 days.

Anxiolytic properties demonstrated in preclinical anxiety models.

Nearly doubles BrdU-positive cells after 4-day treatment.

Mechanism

Synthetic heptapeptide derived from Spadin positions 22-28, functioning as potent TREK-1 antagonist with selectivity and duration versus parent compound. Primary research focus on rapid antidepressant effects.

Selectively blocks TREK-1 potassium channels (IC50: 0.12 nM). serotonin neurotransmission in dorsal raphe nucleus, triggering CREB activation and hippocampal neurogenesis.

Primary research focus with rapid effects in behavioral models within 4 days.

Research areas

  • Synthetic heptapeptide derived from Spadin positions 22-28, functioning as potent TREK-1 antagonist with selectivity and duration versus parent compound. Primary research focus on rapid antidepressant effects.
  • Selectively blocks TREK-1 potassium channels (IC50: 0.12 nM). serotonin neurotransmission in dorsal raphe nucleus, triggering CREB activation and hippocampal neurogenesis.
  • Primary research focus with rapid effects in behavioral models within 4 days.
  • Anxiolytic properties demonstrated in preclinical anxiety models.
  • Nearly doubles BrdU-positive cells after 4-day treatment.

Research notes

  • No effects on TREK-2, TRAAK, TASK-1 channels observed
  • No cardiac dysfunction or seizures in preclinical studies
  • Serotonin syndrome signs
  • Severe persistent headaches
  • Severe mood changes or suicidal ideation

Questions

How much more potent is PE-22-28 compared to full-length Spadin?

PE-22-28 is dramatically more potent—roughly 300-500x more potent than full-length Spadin. This extraordinary difference comes from being a shortened fragment that better mimics the active site. The result is an IC50 of just 0.12 nM for TREK-1 inhibition versus 40-60 nM for Spadin.

Is PE-22-28 safe to combine with SSRIs?

Caution is advised. Both PE-22-28 and SSRIs serotonin. While the interactions section says to 'monitor,' combining them theoretically increases serotonin syndrome risk. Start carefully with your healthcare provider's supervision if considering this combination. Never combine with MAOIs.